cardiology-nutrition-health-2018-posters-abstracts

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Journal of Nutrition and Human Health | Volume 2

October 29-30, 2018 | London, UK

Joint Event

Nutrition and Fitness

International Conference on

World Congress on

Card i o l ogy

Double- chambered left ventricle and abnormal papillary muscle formation

Yassmin Hanfi

University of Paris 7, UK

apillary muscles develop separately from mitral valve

leaflet and chordae. Where papillary muscles origin from

myocardial ridge of the anterior wall and to the posterior wall

of the left ventricle, Chordae and mitral valve leaflets origin

from a cushion tissue. The myocardial ridge gradually loosens

from the ventricular wall and meanwhile the cushion tissue

transforms into leaflet and chordae1. Abnormality in the

development of papillary muscle could be responsible of a

rare form of double chamber LV. The double chamber LV can

be misdiagnosed as aneurysm or rupture LV. Identification

of such form of double chamber LV would have an impact

for the patient management care. Here is a case report of

double chamber LV with variable presentation as previously

published. Case report 17 years oldmale patient with a recent

history of palpitation not related to exertion. Normal ECG

findings. A transthoracic echocardiogram revealed mildly

dilated left ventricle with abnormal trabeculated appearance

and papillary muscle apparatus forming double chamber LV

with mild obstruction. Cardiovascular magnetic resonance

(CMR) cine images showed a well formed compacted

myocardial layer with normal systolic thickening excluding

the LV non compaction pathology. Although confirmed an

anomalous muscular bridge opposite to the normally formed

anterolateral (AL) papillary muscle causing partial division

of the LV in two chambers without significant obstruction

at rest (Panels A, B, D). Peak recorded velocity <1m/s by

velocity mapping at rest (Panel E). While the posteromedial

papillary muscle is abnormally hypoplastic and heavily

fragmented (Panels G, H). The mitral valve found to be with

mild bellowing of its anterior leaflet with no significant

regurgitation. late gadolinium enhancement phase at the

abnormally hypoplastic posteromedian papillary muscle.