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allied
academies
August 16-17, 2018 | Copenhagen, Denmark
Dementia and Alzheimer ’s Disease
10
th
World congress on
Journal of Neurology and Neurorehabilitation Research | Volume: 3
Alzheimer’s cognitive impairment can be recovered by decreasing homocysteic acid in blood
Tohru Hasegawa
Saga Woman Junior College, Japan
R
ecent big two clinical trials of amyloid beta treatment for
Alzheimer’s disease are all failed to recover the cognitive
impairment, it has forcedus to reconsider the central hypothesis
of amyloid pathogen for Alzheimer’s disease. We recently
published that human Alzheimer’s patients showed the strong
relationship between Mini Mental State Examination (MMSE)
scores and blood homocysteic acid (HA) level. 6 AD patients (all
female: age 77, 82, 86, 87, 91, 91) were given green tea powder
1g at every meal for 2 months. Their blood HA level and MMSE
score weremeasured before and after taking green tea powder.
The relationship between blood HA level change and MMSE
score changewas investigated. The strong statistically significant
negativerelationshipbetweenbloodHA level changeandMMSE
score change: r=-0.96, p=0.00018, n=6. From our observation,
it showed that blood HA level change induced MMSE score
change, that is, Alzheimer’s cognitive ability was controlled
by blood HA level. Now we can present that some healthy
food, that is named HBF, can recovered 100% Alzheimer’s
cognitive impairment by the decreasing the homocysteic acid
in a peripheral blood. Now we have made a relative large open
trial of AD patients. 91 patients were enrolled. Their cognitive
recovery was measured by NM scale (New Clinical Scale for
Rating of Mental States). All patients who took HBF showed
the recovery of their behaviors. From this open-trial of HBF, (1)
Alzheimer’s cognitive impairment could be recovered at even
endstage.(2)Alzheimer’sdiseaseisinducedbyhomocysteicacid.
e:
tohru.hasegawa@gmail.com