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Journal of Hematology and Blood Disorder | Volume 2
allied
academies
August 23-24, 2018 | London, UK
Hematology and Oncology
2
nd
International Conference on
W
ith modern medical developments various malignant
disorders such as leukemia or solid organ cancers, have
been transformed from acute life-threatening into chronic
diseases. This transition continues with the growing number
of novel agents that become available to treat diseases from
chronic leukemias to breast cancer, or carcinoma of the lung.
Patients are certainly aware of that, and a frequent question is:
“Well, doctor, I know I have already received two treatments
and have not responded. What are we going to do if this next
treatment is not going to work?” In fact, it happens that you
treat patients and basically have conveyed the message that we
are at the end of the road, the patient considering transition to
palliative or hospice care, when a new study is published with
yet another agent, opening the door again, basically returning
the patient from hospice to active therapy. These situations
come with considerable psychological stress. In addition,
however, the cost of this type of management to the health
care system is phenomenal. Some groups have admonished
physicians to exert some financial stewardship. Others have
argued that we cannot withhold treatment if such treatment
is available. Do we need a new set of ethical rules? No one
likes to set priorities or ration care. Discussions within the
medical community alone will not lead to substantial change.
We must have a conversation within the society at large.
Speaker Biography
H Joachim Deeg completed his MD in Wilhelms Universitaet, Germany. Presently
he is working as a Professor of medicine in the University of Washington. He is
also a member of the Fred Hutchinson Cancer Research Center. He is also a visiting
professor at Carl Carus University, Dresden, Germany. His research interests are
Pathophysiology, genetics and epigenetics of MDS (role of transcription factors
in regulation) Inflammatory responses and GVHD (effects of alpha1 anti-trypsin
[AAT]), Separation of GVHD and GVL effects by AAT, Iron and allogeneic responses.
e:
jdeeg@frehutch.orgH Joachim Deeg
University of Washington, USA
When do we stop? Modern sequential therapies for hematologic malignancies and
end of life questions