Journal of Pain Management and Therapy

All submissions of the EM system will be redirected to Online Manuscript Submission System. Authors are requested to submit articles directly to Online Manuscript Submission System of respective journal.
Reach Us +1 (202) 780-3397

Opinion Article - Journal of Pain Management and Therapy (2024) Volume 8, Issue 1

The intricacies of visceral pain silent suffering

Katherine Mark*

Department of Osteoarthritis, Wake Forest University School of Medicine, United States

*Corresponding Author:
Katherine Mark
Department of Osteoarthritis, Wake Forest University School of Medicine, United States
Wake Forest University School of Medicine
United States
E-mail:katherine@mark.edu

Received:25-Dec-2023, Manuscript No. AAPMT-24-129474; Editor assigned: 28-Dec-2023, PreQC No. AAPMT-24-129474(PQ); Reviewed:11-Jan-2024, QC No. AAPMT-24-129474; Revised:16-Jan-2024, Manuscript No. AAPMT-24-129474(R); Published:22-Jan-2024, DOI:10.35841/aapmt-8.1.186

Citation: Mark K. The intricacies of visceral pain silent suffering. J Pain Manage Ther. 2024;8(1):186

Visit for more related articles at Journal of Pain Management and Therapy

Introduction

Pain, often described as the body's alarm system, serves as a warning signal for potential harm or injury. While most people are familiar with the sensation of pain associated with a cut, burn, or bruise, there exists another type of pain that is less recognized and often misunderstood: visceral pain. Unlike somatic pain, which originates from the skin, muscles, or joints, visceral pain arises from the internal organs of the body, presenting unique challenges in diagnosis, management, and treatment. In this article, we delve into the complexities of visceral pain, shedding light on its underlying mechanisms, common causes, and clinical implications[1, 2].

Visceral pain refers to pain that originates from the internal organs, such as the gastrointestinal tract, urinary bladder, reproductive organs, and thoracic and abdominal organs. Unlike somatic pain, which is often well-localized and easily identifiable, visceral pain tends to be diffuse, poorly localized, and accompanied by a deep, dull, or cramping sensation. This characteristic nature of visceral pain can make it challenging to pinpoint the exact source and diagnose the underlying cause[3].

Mechanism

The mechanisms underlying visceral pain are complex and multifaceted, involving a combination of sensory, neural, and inflammatory processes. Unlike somatic pain, which is mediated by well-defined pain receptors (nociceptors) in the skin and muscles, visceral pain arises from nociceptors located within the walls of the internal organs. These nociceptors detect mechanical distension, chemical irritation, inflammation, and ischemia (lack of blood flow) within the organs, triggering pain signals that are transmitted to the brain via visceral afferent nerves[4, 5].

Visceral pain can arise from a wide range of conditions affecting the internal organs, including Gastrointestinal Disorders Conditions such as gastroesophageal reflux disease, peptic ulcers, inflammatory bowel disease, and irritable bowel syndrome can cause visceral pain due to inflammation, distension, or spasms of the gastrointestinal tract[6].

Disorders of the urinary tract, such as Urinary Tract Infections (UTIs), kidney stones, and interstitial cystitis, can lead to visceral pain in the lower abdomen and pelvis. Conditions affecting the female reproductive organs, such as ovarian cysts, endometriosis, pelvic inflammatory disease (PID), and uterine fibroids, can cause visceral pain localized to the pelvis and lower abdomen. Conditions such as pneumonia, pleurisy, and pericarditis can cause visceral pain in the chest due to inflammation or irritation of the thoracic organs[7].

Diagnosing and managing visceral pain can be challenging due to its diffuse nature and varied underlying causes. A thorough medical history, physical examination, laboratory tests, imaging studies (such as ultrasound, CT scans, or MRI), and invasive procedures (such as endoscopy or cystoscopy) may be necessary to identify the underlying cause[8].

Treatment of visceral pain depends on the underlying condition but may include, medications to alleviate pain, inflammation, or spasms, Dietary modifications to reduce gastrointestinal irritation or symptoms, Physical therapy to improve pelvic floor muscle function or posture, Behavioural therapies to address psychological factors contributing to pain perception, Invasive procedures or surgery to address structural abnormalities or underlying pathology[9].

Visceral pain, though often overshadowed by its more well-known counterpart, somatic pain, represents a significant source of discomfort and disability for many individuals. By understanding the mechanisms, common causes, and clinical implications of visceral pain, healthcare providers can better diagnose and manage this complex condition, ultimately improving the quality of life for those affected. Through a multidisciplinary approach that addresses both physical and psychological aspects of pain, individuals living with visceral pain can find relief and regain control over their lives[10].

References

  1. Abelli L, Conte B, Somma V, et al. A method for studying pain arising from the urinary bladder in conscious, freely-moving rats. J Urol. 1989;141(1):148-51.
  2. Indexed at, Google Scholar, Cross Ref

  3. Akbar A, Yiangou Y, Facer P, et al. Increased capsaicin receptor TRPV1-expressing sensory fibres in irritable bowel syndrome and their correlation with abdominal pain. Gut. 2008;57(7):923-9.
  4. Indexed at, Google Scholar, Cross Ref

  5. Al–Chaer ED, Kawasaki M, Pasricha PJ. A new model of chronic visceral hypersensitivity in adult rats induced by colon irritation during postnatal development. Gastroenterology. 2000;119(5):1276-85.
  6. Indexed at, Google Scholar, Cross Ref

  7. Aldskogius H, Elfvin LG, Forsman CA. Primary sensory afferents in the inferior mesenteric ganglion and related nerves of the guinea pig: An experimental study with anterogradely transported wheat germ agglutinin-horseradish peroxidase conjugate. J Auton Neurosci Sys. 1986;15(2):179-90.
  8. Indexed at, Google Scholar, Cross Ref

  9. Anand KJ. Clinical importance of pain and stress in preterm neonates. Neonatology. 1998;73(1):1-9.
  10. Indexed at, Google Scholar, Cross Ref

  11. Anand KJ, Coskun V, Thrivikraman KV, et al. Long-term behavioral effects of repetitive pain in neonatal rat pups. Physiol Behav. 1999;66(4):627-37.
  12. Indexed at, Google Scholar, Cross Ref

  13. Anand KJ, Runeson B, Jacobson B. Gastric suction at birth associated with long-term risk for functional intestinal disorders in later life. J Paediatr. 2004;144(4):449-54.
  14. Indexed at, Google Scholar, Cross Ref

  15. Apostolidis A, Brady CM, Yiangou Y, et al. Capsaicin receptor TRPV1 in urothelium of neurogenic human bladders and effect of intravesical resiniferatoxin. Urology. 2005;65(2):400-5.
  16. Indexed at, Google Scholar, Cross Ref

  17. Applebaum AE, Vance WH, Coggeshall RE. Segmental localization of sensory cells that innervate the bladder. J Comp Neurol. 1980;192(2):203-9.
  18. Indexed at, Google Scholar, Cross Ref

  19. Bahns E, Ernsberger U, Janig W, et al. Functional characteristics of lumbar visceral afferent fibres from the urinary bladder and the urethra in the cat. Pflugers Archiv. 1986;407:510-8.
  20. Indexed at, Google Scholar, Cross Ref

Get the App