Mini Review - Journal of Oral Medicine and Surgery (2022) Volume 5, Issue 4

A study of oral lichen planus and periodontal maintenance in the treatment of autoimmune diseases.

Guillaume Monet^*

Department of Health Sciences, University of Bordeaux, Bordeaux, France

*Corresponding Author:

Guillaume Monet
Department of Health Sciences
University of Bordeaux, Bordeaux, France
E-mail: monet.guillaume@u-bordeaux.fr

Received: 28- June-2022, Manuscript No. AAOMT-22-68490; Editor assigned: 30- June-2022, PreQC No.AAOMT-22-68490(PQ); Reviewed: 15-July-2022, QC No.AAOMT-22-68490; Revised: 18-July-2022, Manuscript No.AAOMT-22-68490(R); Published: 26-July-2022, DOI: 10.35841/aaomt-5.4.116

Citation: Monet G. A study of oral lichen planus and periodontal maintenance in the treatment of autoimmune diseases. J Oral Med Surg. 2022;5(4):116

Oral lichen planus (OLP) may be a unremitting immune system, mucocutaneous infection that influences the verbal mucosa as well as the skin, genital mucosa, scalp, and nails. It is one of the foremost common dermatological maladies displaying within the in the within the verbal depression. An immunemediated pathogenesis is recognized in lichen planus, in spite of the fact that the precise etiology is obscure. The illness most commonly influences middle-aged females. It is occasionally found in children, with a predominance of approximately 0.03%, and reports of this are rare within the writing. The erosive and atrophic shapes of OLP are less common; however they are more likely to cause symptoms. OLP is the target of much contention, particularly in connection to its potential for threat. Hence, it is vital for clinicians to preserve a tall file of doubt for all intraoral lichenoid injuries. Occasional follow-up of all patients with OLP is prescribed [1].

Oral lichen planus (OLP) may be a or maybe common illness within the middle-aged and elderly populaces. It encompasses a predominance of around 0.5% to 2%, with a female-to-male proportion of roughly 2:1.1. The detailed predominance rates within the Indian populace are 2.6%. In differentiate, oral lichen planus in childhood (OLPc) is uncommon, and as it were a number of reports are accessible within the writing. OLP is classified into reticular, erosive, atrophic, and bullous types. The erosive and reticular shapes are the foremost common sorts and display as papules and plaques with interlap white keratotic lines (Wickham striae) with an erythematous border. The higher recurrence of erosive OLP compared to the reticular and atrophic shapes, as already watched by Silverman, likely the result of the symptomatic nature of this injury, which frequently prompts an assessment visit. The event of erosive OLP limited to the gingival mucosa is characterized by the nearness of diffuse erythematous zones that will or may not be mixed with desquamative and ulcerated foci. The injuries may happen taking after the gingival diagram, and hyperkeratotic emanating straie can be found at the outskirts of the erosive locale. When erosive OLP includes the joined gingival tissue, it is called desquamative gingivitis. The injuries of erosive OLP move over time and tend to be multifocal [2,3].

These variations may happen together in one understanding or may change one to another. The injuries are found (in decreasing recurrence) on the buccal mucosa (frequently symmetrical), horizontal edges of the tongue, gingiva, lips, and palatum durum. Though cutaneous lichen planus is selflimiting, OLP is incessant, seldom experiences unconstrained reduction, and may be a potential source of critical morbidity. Family history of lichen planus is more commonly positive in patients with lichen planus in childhood than in adulthood. The etiology of lichen planus remains dubious, but numerous variables have been involved. Such components incorporate hereditary inclination, infective operators, systemic illnesses, graft-versus-host infection (GVHD), sedate responses, vitamin lacks, and extreme touchiness to dental materials. Lichen planus has been related with a few auto-immune infections, counting lupus erythematosus, pemphigus, Sjogren’s disorder, and immune system liver disease. The pathogenesis of lichen planus isn't totally caught on, but a T lymphocyte invade proposes cell-mediated immunological harm to the epithelium. Altered Langerhans’ cells and keratinocytes may trigger a safe reaction and the enlistment of T lymphocytes caused by expression of cell-surface grip molecules. Both CD4 (aide) and CD8 (cytotoxic) cells are show, but expanding numbers and enactment of the CD8 cells are thought to contribute to the characteristic harm to the basal epithelium [4].

It is critical for all clinicians to be mindful of its clinical introductions and administration, since OLP is one of the foremost common mucosal conditions affecting the verbal depression. The foremost broadly acknowledged treatment for OLP is topical corticosteroids. Elective medicines incorporate retinoids, cyclosporine, tacrolimus, surgery, and carbon dioxide (CO2) laser. This report highlights the utilize of topical and systemic corticosteroids and retinoids for the administration of erosive gingival lichen planus in a youthful; it too incorporates a survey of current writing on this condition. Lichen planus was to begin with depicted within the writing by Eramus Wilson in 18692 as predominately a malady of the middle-aged or more seasoned. There's restricted writing accessible announcing the events of verbal lichen planus in children. Childhood lichen planus has been archived as a complication of hepatitis B infection (HPV) inoculation, where the recombinant proteins of the HBV vaccine—specifically the viral S epitope may trigger a cell-mediated immune system reaction focused on at keratinocytes, giving rise to a lichenoid reaction.It is additionally found in affiliation with inclining conditions such as graft-versus-host illness (GVHD) and constant dynamic hepatitis C. detailed verbal lichen planus inclusion in one understanding influenced by immune system persistent dynamic hepatitis, detailed oral injuries in one quiet after HBV immunization [5].

References

1. Alsarraf A, Mehta K, Khzam N. The gingival oral lichen planus: A periodontal-oral medicine approach. Case Reports in Dentistry. 2019;4659134.

Indexed at, Google Scholar, Cross Ref

2. Mignogna MD, Russo LL, Fedele S. Gingival involvement of oral lichen planus in a series of 700 patients. J Clin Periodontol. 2005;32(10):1029-33.

Indexed at, Google Scholar, Cross Ref

3. Salgado DS, Jeremias F, Capela MV, et al. Plaque control improves the painful symptoms of oral lichen planus gingival lesions. A short?term study. J Oral Pathol Med. 2013;42(10):728-32.

Indexed at, Google Scholar, Cross Ref

4. Gowri Pendyala MD, Saurabh Joshi MD, Jithendra Kalburge MD, et al. Oral Lichen Planus: A Report and Review of an Autoimmune-Mediated Condition in Gingiva. Compend. 2012;33(8).

Indexed at, Google Scholar

5. Roopashree MR, Gondhalekar RV, Shashikanth MC,et al. Pathogenesis of oral lichen planus–a review. J Oral Pathol Med. 2010;39(10):729-34.

Indexed at, Google Scholar, Cross Ref